The binding of MSL2 or one of the mutants to three high-affinity sites CES11D1, roX1, and Set2-H or to a control locus that has no MRE sequences Set from two independent biological replicates is shown side by side and expressed as percentage of input. Functional integration of the histone acetyltransferase MOF into the dosage compensation complex. These mutations did not grossly perturb the structure, as all mutant proteins still displayed a single folded conformation in HSQC spectra Fig. A single small CXC domain offers only limited binding specificity and affinity. Residues — were not all modeled in some molecules due to weak electron density. The fitting curves were displayed in Supplemental Figure S6.
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